山东大学教师主页曾涛论文成果中文主页

个人信息

曾涛

性别：男
在职信息：在职

个人信息 Personal information

在职信息：在职所在单位：公共卫生学院入职时间：2010-07-16 所属院系：齐鲁医学院 , 公共卫生学院 , 公共卫生学院 , 公共卫生学院 , 公共卫生学院

论文成果

中文主页 - 科学研究 - 论文成果

[1] 寇蕊蕊. 毒理学案例库深度融合PBL实验教学模式探索. 卫生职业教育, 43, 93, 2025.
[2] . Sensitivity and stability of Balb/c, C57BL/6J, and ICR mice to the acute liver injury induced by concanavalin A. Toxicology Research, 14, 1, 2025.
[3] . Allyl methyl disulfide attenuates liver injury induced by concanavalin A by suppressing M1 polarization of macrophages and NLRP3 inflammasome activation. International Immunopharmacology, 149, 114149, 2025.
[4] 寇蕊蕊. 综合创新性实验在毒理学实验教学中的实践评价. 《实验室科学》, 27, 60, 2024.
[5] 张一帆. Chronic di(2-ethylhexyl) phthalate exposure at environmental-relevant doses induces osteoporosis by disturbing the differentiation of bone marrow mesenchymal stem cells. Sci Total Environ, 2024.
[6] 张秀凝. NLRP3 inflammasome activation triggers severe inflammatory liver injury in N, N-dimethylformamide-exposed mice. Science of The Total Environment , 929, 2024.
[7] 张一帆. Chronic di(2-ethylhexyl) phthalate exposure at environmental-relevant doses induces osteoporosis by disturbing the differentiation of bone marrow mesenchymal stem cells. Science of The Total Environment , 914, 2024.
[8] . Allyl methyl disulfide (AMDS) prevents N,N-dimethyl formamide-induced liver damage by suppressing oxidative stress and NLRP3 inflammasome activation. Food Chem Toxicol., 182, 2023.
[9] 高璇. Allyl methyl disulfide ameliorates concanavalin A-induced liver injury by inhibiting M1 polarization of macrophages and NLRP3 inflammasome activation. Gastroenterology, 164, 1432, 2023.
[10] 李萍. 重金属镉暴露致肝纤维化的机制研究进展. 《毒理学杂志》, 37, 54-56, 2023.

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