山东大学教师主页陈丙玺 Paper Publications Home

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Gender:Male
Date of Employment:1980-02

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[1] 邹永新 , 高贵敏 , 陈丙玺 , 刘奇迹 , Yaoqin Gong , 蒋百春 and Yaoqin Gong. Sp1 and CREB regulate basal transcription of the human SNF2L gene.. Biochem Biophys Res Commun., 368, 438, 2008.
[2] 周海滨 , 高贵敏 , 陈丙玺 , 刘奇迹 , 邵常顺 , Yaoqin Gong and Yaoqin Gong. Serum withdrawal up-regulates human SIRT1 gene expression in a p53-dependent manner.. J Cell Mol Med., 13, 4176, 2009.
[3] 蒋百春 , 孙文杰 , 邹永新 , 郭辰虹 , 陈丙玺 , 邵常顺 , Yaoqin Gong and Yaoqin Gong. Lack of Cul4b, an E3 ubiquitin ligase component, leads to embryonic lethality and abnormal placental development.. PLoS ONE., 7, 37070, 2012.
[4] Yaoqin Gong , 邵常顺 , 刘奇迹 , 周海滨 , 李曦 , 张锡宇 , 高贵敏 , 郭辰虹 and 陈丙玺. Resveratrol augments the canonical Wnt signaling pathway in promoting osteoblastic differentiation of multipotent mesenchymal cells.. Exp Cell Res., 315, 2953, 2009.
[5] 张锡宇. A locus for nonspecific X-linked mental retardation mapped to a 22.3 cM region of xp11.3-q22.3. AMERICAN JOURNAL OF MEDICAL GENETICS ..., 129A, 286, 2004.
[6] 邹永新 , 郭辰虹 , 邵常顺 , 蒋百春 , Yaoqin Gong , 陈丙玺 and 孙文杰. Lack of Cul4b, an E3 ubiquitin ligase component, leads to embryonic lethality and abnormal placental development.. PLoS ONE., 7, e37070, 2012.
[7] Yaoqin Gong , 邵常顺 , 邹永新 , 刘奇迹 , 陈丙玺 and 郭辰虹. Characterization of Nuclear Localization Signal in the N Terminus of CUL4B and Its Essential Role in Cyclin E Degradation and Cell Cycle Progression. Journal of biological chemistry, 284, 33320, 2009.
[8] 刘奇迹 , 陈丙玺 , 郭辰虹 , Yaoqin Gong and 邹永新. Characterization of Nuclear Localization Signal in the N Terminus of CUL4B and Its Essential Role in Cyclin E Degradation and Cell Cycle Progression. Journal of biological chemistry, 284, 33320, 2009.
[9] Yaoqin Gong , 周海滨 , 李曦 , 张锡宇 , 高贵敏 , 郭辰虹 , 陈丙玺 , 刘奇迹 and 邵常顺. Resveratrol augments the canonical Wnt signaling pathway in promoting osteoblastic differentiation of multipotent mesenchymal cells.. Exp Cell Res., 315, 2953, 2009.
[10] Yaoqin Gong , 邵常顺 , 孙文杰 , 邹永新 , 郭辰虹 , 陈丙玺 and 蒋百春. Lack of Cul4b, an E3 ubiquitin ligase component, leads to embryonic lethality and abnormal placental development.. PLoS ONE., 7, e37070, 2012.

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