张艳
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Gender:Female
Paper Publications
- [1] 刘素侠. 互补于hTERT关键区段的反义RNA抑制肝癌细胞研究. 基础医学与临床, 26, 381, 2006.
- [2] . Tubular MYDGF Slows Progression of Chronic Kidney Disease by Maintaining Mitochondrial Homeostasis. Advanced Science, 2024.
- [3] 贾蒙. Targeting RNA oxidation by ISG20-mediated degradation is a potential therapeutic strategy for acute kidney injury. MOLECULAR THERAPY, 31, 3034, 2023.
- [4] 左富文. CCDC92 promotes podocyte injury by regulating PA28α/ABCA1/cholesterol efflux axis in type 2 diabetic mice. ACTA PHARMACOLOGICA SINICA, 2024.
- [5] 左富文. CCDC92 deficiency ameliorates podocyte lipotoxicity in diabetic kidney disease. METABOLISM-CLINICAL AND EXPERIMENTAL, 150, 2023.
- [6] 张杨. HDAC9-mediated epithelial cell cycle arrest in G2/M contributes to kidney fibrosis in male mice. NATURE COMMUNICATIONS, 2023.
- [7] 刘素侠. 靶向于端粒酶调控区反义RNA腺病毒载体的构建. 《山东大学学报》（医学版）, 43, 667, 2005.
- [8] 崔晓阳. GPR87 promotes renal tubulointerstitial fibrosis by accelerating glycolysis and mitochondrial injury. Free Radical Biology & Medicine, 58, 2022.
- [9] . GPR97 deficiency ameliorates renal interstitial fibrosis in mouse hypertensive nephropathy. ACTA PHARMACOLOGICA SINICA, 2023.
- [10] 孙玉. JAML promotes acute kidney injury mainly through a macrophage-dependent mechanism.. JCI Insight, 2022.