朱法良山东大学主页平台管理系统--中文主页

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朱法良

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讲师

性别：男

毕业院校：山东大学

学历：研究生(博士)毕业

学位：医学博士学位

在职信息：在职

所在单位：基础医学院

入职时间：2000-07-01

学术荣誉：

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当前位置中文主页 >> 科学研究 >> 论文成果

[1] 马湉. Th17 cells and IL-17 are involved in the disruption of vulnerable plaques triggered by short-term co. Cell Mol Immunol, 10, 338, 2013.
[2] 朱法良. Blockade of CD28 by a synthetical peptoid inhibits T-cell proliferation and attenuates graft-versus-. Cellular and Molecular Immunology, 7, 133, 2010.
[3] . IL-37d enhances COP1-mediated C/EBPβ degradation to suppress spontaneous neutrophil migration and t. Cell Reports, 43, 2024.
[4] 郭亚新. D-arabinose acts as antidepressant by activating the ACSS2-PPARγ/TFEB axis and CRTC1 transcription. Pharmacological Research, 202, 2024.
[5] . ACSS2 controls PPARγ activity homeostasis to potentiate adipose-tissue plasticity. Cell Death and Differentiation, 2024.
[6] 陈诺. IL-37d suppresses Rheb-mTORC1 axis independently of TCS2 to alleviate alcoholic liver disease. Communications Biology , 7, 2024.
[7] 陈诺. Brain Short-Chain Fatty Acids Induce ACSS2 to Ameliorate Depressive-Like Behavior via PPARγ-TPH2 Ax. Research, 7, 2024.
[8] 周子歆. FGF21 alleviates adipose stem cell senescence via CD90 glycosylation-dependent glucose influx in rem. Redox Biology, 67, 2023.
[9] . Hepatocyte TIPE2 is a fasting-induced Raf-1 inactivator that drives hepatic gluconeogenesis to maint. METABOLISM-CLINICAL AND EXPERIMENTAL, 148, 2023.
[10] 赵明. D-mannose acts as a V-ATPase inhibitor to suppress inflammatory cytokines generation and bacterial k. Molecular Immunology, 162, 84-94, 2023.
[11] . D-mannose is a rapid inducer of ACSS2 to trigger rapid and long-lasting antidepressant responses thr. Translational psychiatry, 13, 2023.
[12] Li, haiyan. Reduced expression of PDCD5 is associated with high-grade astrocytic gliomas. Ocology Report, 20, 573-579, 2008.
[13] 高琦. A Critical Function of Th17 Proinflammatory Cells in the Development of Atherosclerotic Plaque in Mi. J Immunol , 185, 5820-5827, 2010.
[14] 宋兴国. Tumor suppressor gene PDCD4 negatively regulates autophagy by inhibiting the expression of autophagy. Autophagy, 9, 743-755, 2013.
[15] 江杨. Deficiency of programmed cell death 4 affects the balance of T cell subsets in hyperlipidemic mice. Molecular Immunology, 112, 387, 2019.
[16] 刘素侠. 靶向于端粒酶调控区反义RNA腺病毒载体的构建. 《山东大学学报》（医学版）, 43, 667, 2005.
[17] 周子歆. CX3CR1hi macrophages sustain metabolic adaptation by relieving adipose-derived stem cell senescence . Cell Reports, 42, 112424, 2023.
[18] . IL-37 isoform D acts as an inhibitor of soluble ST2 to boost type 2 immune homeostasis in white adip. CELL DEATH DISCOVERY, 8, 163, 2022.
[19] 李文. TNFAIP8L2/TIPE2 impairs autolysosome reformation via modulating the RAC1-MTORC1 axis. Autophagy, 2021.
[20] 陈晓彤. Programmed cell death 4 modulates lysosomal function by inhibiting TFEB translation. Cell Death and Differentiation, 2021.

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