Professor
山东大学公共卫生学院研究员，硕士/博士研究生导师，山东大学“齐鲁青年学者”，2022年入选“第七届中国科协青年人才托举工程”。主要从事环境及职业性有害因素（电离辐射）所致DNA损伤应答与基因组稳定性调控机制，胃肠道疾病发病机理、肿瘤发生及信号转导机制的研究。相关研究成果在 PNAS，Nucleic Acids Res，Cell Death Dis.等国际知名学术期刊发表。 招生方向：学术型（劳动卫生学）；专业型（公共卫生-环境/职业与健康）。Email: chaodong@sdu.edu.cn 欢迎邮件咨询。 More+
Paper Publications
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[1] 陈晨. HDAC inhibitor HPTA initiates anti-tumor response by CXCL9/10-recruited CXCR3+CD4+T cells against PAHs carcinogenicity. Food and chemical toxicology, 2023.
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[2] 苏本玉. VPA mediates bidirectional regulation of cell cycle progression through the PPP2R2A-Chk1 signaling axis in response to HU. Cell Death & Disease, 14, 2023.
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[3] 贾贝迪. Global research trends in radiotherapy for breast cancer: a systematic bibliometric analysis. JAPANESE JOURNAL OF RADIOLOGY, 2023.
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[4] Yanling Yao , Chen Chen , Zuchao Cai , Guochao Liu , Chenxia Ding , David Lim , Chao Dong and Zhihui Feng. Screen identifies fasudil as a radioprotector on human fibroblasts. Toxicology Research, 2022.
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[5] Chao Dong. Valproic acid counteracts polycyclic aromatic hydrocarbons (PAHs)-induced tumorigenic effects by regulating the polarization of macrophages Author links open overlay panel. Ecotoxicology and Environmental Safety, 241, 2022.
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[6] 张怡莎. Global Research Trends in Radiotherapy for Gliomas: A Systematic Bibliometric Analysis. World Neurosurgery, 161, 355, 2022.
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[7] David Lim. Valproic Acid-Like Compounds Enhance and Prolong the Radiotherapy Effect on Breast Cancer by Activating and Maintaining Anti-Tumor Immune Function, Frontiers in Immunology. Frontiers in Immunology, 2021.
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[8] 刘国超. The Valproate Mediates Radio-Bidirectional Regulation Through RFWD3-Dependent Ubiquitination on Rad51. FRONTIERS IN ONCOLOGY, 11, 2021.
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[9] 董超. A DYRK1B-dependent pathway suppresses rDNA transcription in response to DNA damage. Nucleic Acids Research, 49, 2021.
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[10] 彭君璇. The intervention of valproic acid on the tumorigenesis induced by an environmental carcinogen of PAHs. Toxicology Research, 9, 609, 2020.
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[11] 董超. Screen identifies DYRK1B network as mediator of transcription repression on damaged chromatin. PNAS, 117, 2020.
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[12] 董超. RNF169 limits 53BP1 deposition at DSBs to stimulate single-strand annealing repair. PNAS, 115, 2018.
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[13] 董超. p53 suppresses hyper-recombination by modulating BRCA1 function. DNA repair, 33, 60, 2015.