|
个人信息Personal Information
教授 博士生导师 硕士生导师
性别：男
毕业院校：香港大学
学历：研究生(博士)毕业
学位：博士生
在职信息：在职
所在单位：高等医学研究院
入职时间：2019-04-16
学科：免疫学
办公地点：山东大学千佛山校区10号楼，高等医学研究院
电子邮箱：wphlab@163.com
扫描关注
- [1] 武专昌. Peptide targeting the interaction of S protein cysteine-rich domain with Ezrin restricts pan-coronavirus infection. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 8, 2023.
- [2] . SARS-CoV-2 NSP8 suppresses type I and III IFN responses by modulating the RIG-I/MDA5, TRIF, and STING signaling pathways. Journal of Medical Virology, 2023.
- [3] . SARS-CoV-2 NSP7 inhibits type I and III IFN production by targeting the RIG-I/MDA5, TRIF, and STING signaling pathways. Journal of Medical Virology, 2023.
- [4] . SARS-CoV-2 NSP5 and N protein counteract the RIG-I signaling pathway by suppressing the formation of stress granules. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 2022.
- [5] . SARS-CoV-2 ORF10 antagonizes STING-dependent interferon activation and autophagy.. Journal of Medical Virology, 2022.
- [6] Wu, Feima. Generation of WAe001-A-58 human embryonic stem cell line with inducible expression of the SARS-CoV-2 nucleocapsid protein.. STEM CELL RESEARCH, 53, 102197, 2021.
- [7] 郑义. SARS-CoV-2 ORF10 antagonizes STING-dependent interferon activation and autophagy. Journal of Medical Virology, 2022.
- [8] 郑义. SARS-CoV-2 NSP5 and N protein counteract the RIG-I signaling pathway by suppressing the formation of stress granules. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 7, 2022.
- [9] Zhang, Yuehui. An antibody-based proximity labeling map reveals mechanisms of SARS-CoV-2 inhibition of antiviral immunity. CELL CHEMICAL BIOLOGY, 29, 5, 2022.
- [10] Hu, Wei. Mechanical activation of spike fosters SARS-CoV-2 viral infection. 细胞研究（英文版）, 2021.
- [11] Wu, Feima. Generation of WAe001-A-58 human embryonic stem cell line with inducible expression of the SARS-CoV-2 nucleocapsid protein. STEM CELL RESEARCH, 53, 2021.
- [12] 武专昌. Palmitoylation of SARS-CoV-2 S protein is essential for viral infectivity. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 6, 2021.
- [13] 韩璐璐. SARS-CoV-2 ORF9b antagonizes type I and III interferons by targeting multiple components of the RIG-I/MDA-5-MAVS, TLR3-TRIF, and cGAS-STING signaling pathways. Journal of Medical Virology, 93, 5376, 2021.
- [14] Wu, Yaoxing. Main protease of SARS-CoV-2 serves as a bifunctional molecule in restricting type I interferon antiviral signaling. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 5, 2020.
- [15] 庄梦玮. Increasing host cellular receptor-angiotensin-converting enzyme 2 expression by coronavirus may facilitate 2019-nCoV (or SARS-CoV-2) infection. Journal of Medical Virology, 92, 2693, 2020.
- [16] Chen, Hui. Liquid-liquid phase separation by SARS-CoV-2 nucleocapsid protein and RNA. 细胞研究（英文版）, 30, 1143, 2020.
- [17] Jiang, He-wei. SARS-CoV-2 Orf9b suppresses type I interferon responses by targeting TOM70. CELLULAR & MOLECULAR IMMUNOLOGY, 17, 998, 2020.
- [18] 郑义. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) membrane (M) protein inhibits type I and III interferon production by targeting RIG-I/MDA-5 signaling. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 5, 2020.
- [19] 张静. A systemic and molecular study of subcellular localization of SARS-CoV-2 proteins. SIGNAL TRANSDUCTION AND TARGETED THERAPY, 5, 2020.
- [20] Wang, Bin. Long-term coexistence of SARS-CoV-2 with antibody response in COVID-19 patients. Journal of Medical Virology, 92, 1684, 2020.